How long after fetal demise does the uterus become infected?

There is no fixed timeframe for uterine infection following fetal demise; the key factor lies in how promptly embryonic tissue is expelled. If the embryonic tissue is naturally or surgically removed within a short period, the risk of infection is low. However, if the tissue remains in the uterus for more than two weeks, the likelihood of infection increases significantly, warranting heightened vigilance. Detailed analysis is as follows:

The retention of embryonic tissue in the uterus is the primary cause of infection. After fetal development stops, the embryonic tissue gradually necrotizes and becomes a breeding ground for bacteria. If the retained tissue is present only briefly and the pregnant woman has strong immunity, infection symptoms may not appear immediately. However, as retention time extends, bacterial proliferation intensifies, potentially leading to endometritis.

Infection occurrence also varies with individual differences. Pregnant women with weakened immune systems or a history of gynecological inflammation may develop infections more rapidly when embryonic tissue is retained, possibly exhibiting symptoms such as fever, abdominal pain, and abnormal vaginal discharge within about one week. In contrast, women in better health may experience delayed onset of infection, but once retention exceeds two weeks, the infection risk rises substantially.

It is important to closely monitor vaginal bleeding and the odor of vaginal discharge in daily life, seeking prompt medical attention upon noticing any abnormalities. After fetal demise, timely medical consultation is essential to remove residual tissue via medication or surgery. Post-procedure, maintaining external genital hygiene, avoiding tub baths and sexual intercourse can help reduce the risk of infection.