How is liver calcification treated?
Dec 03, 2025
Source: Cainiu Health
Dr. Gao Jun
Introduction
In general, hepatic calcification may result from congenital developmental abnormalities, age-related degenerative changes, residual effects after recovery from chronic hepatitis, stimulation by intrahepatic bile duct stones, or scarring during the recovery phase of liver abscess. It is recommended to seek medical attention promptly, identify the underlying cause, and then improve the condition through general management, medication, or other treatments under a doctor's guidance.
In general, liver calcification may result from congenital developmental abnormalities, age-related degenerative changes, residual effects after healing of chronic hepatitis, stimulation from intrahepatic bile duct stones, or scar formation during the recovery phase of liver abscess. It is recommended to seek timely medical evaluation to determine the underlying cause, followed by appropriate management under a doctor's guidance through general measures or medication. Specific causes are analyzed as follows:
1. Congenital developmental abnormality: During embryonic development, abnormal liver cell growth may lead to localized deposition of calcium salts and formation of calcified foci. These are usually asymptomatic and require no special treatment. Regular follow-up with liver ultrasound every six months to one year is advised to monitor any size changes in the calcifications, along with maintaining a regular daily routine.
2. Age-related degenerative change: With aging, reduced metabolic function of hepatocytes can disrupt local calcium metabolism, leading to calcium deposition. It is advisable to consume foods rich in vitamin D and calcium, avoid excessive calcium supplementation, engage in moderate physical activity to enhance metabolism, and undergo regular liver function tests.
3. Residual effect after chronic hepatitis healing: After recovery from chronic hepatitis such as hepatitis B or C, necrotic liver tissue may form scars with subsequent calcium salt deposition. Follow your doctor’s instructions to use medications like entecavir tablets, tenofovir disoproxil fumarate tablets, or silibinin capsules to protect liver function, and avoid alcohol consumption to prevent additional liver strain.
4. Stimulation from intrahepatic bile duct stones: Long-term irritation of the bile duct wall by stones may trigger inflammatory reactions resulting in calcium deposition. Asymptomatic cases should be monitored regularly. For symptomatic cases such as abdominal pain, medications including ursodeoxycholic acid capsules, anti-inflammatory and cholagogue tablets, or cefixime dispersible tablets may be prescribed. In some cases, surgical removal of stones via bile duct incision may be necessary.
5. Scar formation during liver abscess recovery: After resolution of a liver abscess, local fibrosis of tissues may secondarily lead to calcium deposition. If residual inflammation is present, medications such as metronidazole tablets, ceftriaxone sodium injection, or levofloxacin tablets may be used. Regular follow-up is important to confirm lesion stability, and spicy or greasy foods should be avoided.
For daily care, maintain a low-fat diet, avoid hepatotoxic drugs, and adhere to a regular sleep schedule without staying up late. With regular monitoring and proper health management, most liver calcifications do not affect liver function and generally do not require excessive concern.
