Why can't patients with cerebral thrombosis fall asleep at night, and what should be done?

Insomnia in patients with cerebral thrombosis at night may be caused by physiological factors, and timely improvement of the sleep environment can help alleviate discomfort. It could also result from the disease itself or neurasthenia, which require proper medical treatment. Specific analyses are as follows:

1. Physiological Factors

If the sleep environment for a patient with cerebral thrombosis is poor—such as excessive light intensity or loud noise—it may lead to difficulty falling asleep. Timely improvements to the sleep environment should be made to relieve these symptoms.

2. Pathological Factors

1) Effects of the Disease Itself

After developing cerebral thrombosis, brain cells may become damaged, leading to excessive psychological stress and mental tension, which in turn causes insomnia. Treatment under medical guidance may include medications such as enteric-coated aspirin tablets, clopidogrel hydrogen sulfate tablets, and ozagrel sodium injection.

2) Neurasthenia

Prolonged mental stress and high pressure following cerebral thrombosis may trigger neurasthenia, keeping the nervous system in a constant state of tension and making it difficult to fall asleep. Under medical supervision, medications such as venlafaxine hydrochloride sustained-release capsules, duloxetine hydrochloride enteric-coated capsules, and paroxetine hydrochloride tablets may be prescribed.

Besides the common factors mentioned above, insomnia may also be caused by depression, cerebral infarction, or coronary atherosclerotic heart disease. When patients with cerebral thrombosis experience insomnia, they should promptly seek medical evaluation and appropriate treatment to promote recovery.